STRENGTH TRAINING HAS LIMITED POTENTIAL
MacDougall, J. D., Ward, G. R., Sale, D. G., & Sutton, J. R. (1977). Biochemical adaptation of human skeletal muscle to heavy resistance training and immobilization. Journal of Applied Physiology, 43, 700-703.
Low-resistance repeated contractions develop significant increases in the capacity of muscle to oxidize pyruvate and fatty acids as a result of both increased mitochondrial enzyme activity and an increase in number and size of mitochondria.
Brief, maximal contractions associated with heavy-resistance strength training require a very high rate of energy production which can be met only by the muscle's high-energy phosphate reserves and to a lesser extent glycolysis.
Although an increase in the total content of the high-energy phosphate pool would not be expected to affect the maximal rate of power output from a muscle, it would increase the total energy available from this source, and thus prolong the time that this rate of power output could be sustained.
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